Disregard Everything I Say

Dosage (oral)
Threshold: 100mg
Light: 100 - 300mg
Common: 300 - 600mg
Strong: 600 - 900mg
Heavy: 900mg +

Duration (oral)
Total duration: 5 - 6 hours
Onset: 1 - 2 hours
Peak: 2 - 3 hours
Come down: 1 - 2 hours
After effects: 2 - 3 hours

3-MeO-PCMo (4-[1-(3-methoxyphenyl)cyclohexyl]morpholine) is a new morpholine analogue of 3-MeO-PCP. It is a dissociative NMDA receptor antagonist and anesthetic drug of the arylcyclohexylamine chemical class with a potency of less than 1/10th of that of 3-MeO-PCP.

This compound induces a state referred to as “dissociative anesthesia” when ingested and is therefore used as a recreational drug. 3-MeO-PCMo has recently become freely available through online research chemical vendors^[1] where it is being sold as a designer drug.

Although very little is known about this compound, similar morpholine analogues of phencyclidine have been researched before.^[2][3]

In terms of its chemistry, 3-MeO-PCMo, or 4-[1-(3-methoxyphenyl)cyclohexyl]morpholine, is classified as an arylcyclohexylamine drug. Ayrlcyclohexylamine drugs are named for their structures which include a cyclohexane ring bound to an aromatic ring along with an amine group at the same location. The aryl substituent of 3-MeO-PCMo is a phenyl ring with a methoxy (CH₃-O-) substituent at R₃, which is bound to a six-membered cyclohexyl ring. Bound at the same location on the cyclohexyl ring R₁ is an amine group which is incorporated into a morpholine ring as R₄. Morpholine is a six-membered heterocyclic ring with an oxygen subsitutent at R₁. 3-MeO-PCMo is a morpholine analogue to 3-MeO-PCP, which lacks an oxygen moiety in its six-membered amine ring (a piperidine ring instead of a morpholine ring).

In terms of its pharmacology, due to the lack of research regarding the substance, all discussion regarding the pharmacology of it is purely based on its structure and subjective effect similarities to other arylcyclohexylamine dissociatives such as 3-MeO-PCP, PCP and MXE. With this in mind, 3-Meo-PCMo is thought to act as an NMDA receptor antagonist. NMDA receptors allow for electrical signals to pass between neurons in the brain and spinal column; for the signals to pass, the receptor must be open. NMDA receptor antagonists close the NMDA receptors by blocking them. This disconnection of neurons leads to loss of feeling, difficulty moving, and eventually the famous “hole.” 

The effects listed below are based upon the subjective effects index and personal experiences of PsychonautWiki contributors. The listed effects will rarely (if ever) occur all at once, but heavier dosages will increase the chances and are more likely to induce a full range of effects.

Physical effects:

The subjective physical effects of 3-MeO-PCMo can be broken down into several components which progressively intensify proportional to dosage. These are described below and generally include:

• Spontaneous tactile sensations - The 3-MeO-PCMo body high is a soft, warm, motionless and pleasurable tingling sensation which is all-encompassing across the body and not location specific to any area.
• Decreased bodily weight - This creates the sensation that the body is floating and has become entirely weightless. This effect is strangely stimulating and encourages physical activities at low to moderate doses by making the body feel light and effortless to move.
• Nausea - It’s worth noting that high dose 3-MeO-PCMo trips can sometimes result in nausea and vomiting at the peak of the trip. For most people, this is surprisingly not as unpleasant as they would initially expect due to the accompanying detachment from the physical senses.
• Motor control loss - A loss of gross and fine motor control alongside of balance and coordination is prevalent within 3-MeO-PCMo and becomes especially strong at higher doses. This means that one should be sitting down before the onset (unless experienced) in case of falling over and injuring oneself.
• Physical euphoria
• Tactile disconnection
• Tactile suppression
• Physical autonomy

Cognitive effects:

The general head space of 3-MeO-PCMo is often described as simplistic and shallow in comparison to that of MXE and ketamine. The specific cognitive effects can be broken down into several separate subcomponents which are listed and described below:

• Depersonalization
• Derealization
• Consciousness disconnection
• Memory suppression (ego death)
• Thought deceleration
• Immersion enhancement
• Information processing suppression
• Time distortion
• Euphoria
• Introspection
• Déjà vu
• Conceptual thinking
• Anxiety suppression
• Disinhibition
• Amnesia

Visual effects:

Suppressions:

This substance does not enhance visual stimuli; instead, it tends to degrade and decrease visual aptitude in a variety of ways which generally include:

• Visual disconnection - This eventually results in the 3-MeO-PCMo equivalent of the famous “k-hole” or, more specifically, holes, spaces and voids alongside of structures.
• Double vision - This component is prevalent at moderate to heavy doses and makes reading impossible unless one closes an eye.
• Pattern recognition suppression - This effect generally occurs at higher doses and makes one unable to recognize and interpret perceivable visual data.
• Visual acuity suppression

Distortions:

• Perspective distortions

Geometry:

The visual geometry found within 3-MeO-PCMo can be described as very dark and bland when compared to that of ketamine or DXM. It often consists of many tiny interlocking and woven lines. It does not extend beyond level 4 geometry and can be comprehensively described through its variations as simplistic in complexity, algorithmic in style, synthetic in feel, unstructured in organization, dimly lit in lighting, multicoloured in scheme, glossy in shading, soft in edges, small in size, slow in speed, smooth in motion, equal in rounded and angular corners, immersive in depth and consistent in intensity.

Auditory effects:

• Suppression
• Distortions
• Hallucinations

Physical health effects, potential addiction and tolerance:

The toxicity and long-term health effects of recreational 3-MeO-PCMo use have not been studied in any scientific context and the exact toxic dose is unknown. This is because 3-MeO-PCMo is a research chemical with very little history of human usage. Anecdotal evidence from people who have tried 3-MeO-PCMo within the psychonaut community suggests that there are no negative health effects attributed to simply trying this drug at low to moderate doses or using it very sparingly (but nothing can be completely guaranteed).

There is a tolerance build up with 3-MeO-PCMo which results in the need to consume increasingly large doses in order to achieve the same level of effects. This should reset to baseline after 1 - 2 weeks. There may be some addictive potential, but this is still unknown.

In terms of its long-term health effects when used repeatedly and with excess for extended periods of time, 3-MeO-PCMo seems to exhibit almost identical bladder and urinary tract problems to those found within ketamine, MXE and other arylcyclohexylamines, but to an even more extreme extent. This is because 3-MeO-PCMo is 50% less potent than that of ketamine which means significantly more of the drug must be consumed in order to achieve the same effects. Symptoms of 3-MeO-PCMo-induced cystitis can become extremely serious and can be described as:

• Urinary frequency - Urinary frequency is the need to empty the bladder every few minutes.
• Urinary urgency - This can be described as a sudden and compelling need to urinate.
• Urinary pressure - This is experienced as a constant sensation of fullness in the bladder that is unrelieved by urination.
• Pelvic and bladder pain - Pain can develop suddenly and severely, particularly as the bladder fills with urine.
• Hematuria - Hematuria is visible blood in the urine.
• Incontinence - This is the leakage of urine.

All of these, however, can easily be avoided by simply not using 3-MeO-PCMo on a daily or even weekly basis and manually limiting one’s usage of the substance.

Legal issues:

3-MeO-PCMo is currently a legal grey area drug worldwide and is freely available through the use of online research chemical vendors. However, this does not guarantee anyone to be immune from legal prosecution should they be found in possession of this substance as the legality is likely to vary from country to country.

Within the U.K. specifically, as 3-MeO-PCMo is the morpholine analogue of 3-MeO-PCP, it is therefore an arylcyclohexylamine (ACH) that is not controlled in the U.K. because a morpholine substituion on the amine is not proscribed by the country’s generic ACH laws.

Conclusion:

3-MeO-PCMo is by far the closest experience to that of MXE or ketamine currently available in the U.K. legal research chemical market. Its absurd lack of potency, however, makes it unusable for regular usage due to its proportionally adverse effects on the bladder.

This substance breakdown was written through collaboration between PsychonautWiki contributors Kaylee, Josikins and Oscarette. The full article is available here.

Dosage (oral)
Threshold: 50 - 75mg
Light: 75 - 110mg
Common: 110 - 150mg
Strong: 150 - 200mg
Heavy: > 200mg +

Duration (oral)
Total duration: 5 - 7 hours
Onset: 10 - 30 minutes

Ephenidine (N-Ethyl-1,2diphenylethylamine) is a dissociative NMDA receptor antagonist and anesthetic drug of the phenethylamine chemical class. It has similar effects to that of the arylcyclohexylamine and morphinan classes of dissociatives.

This compound induces a state referred to as “dissociative anesthesia” and is used as a recreational drug. Very little is known about this substance, but it has recently become freely available through online research chemical vendors where it is being sold as a designer drug. Due to the lack of research regarding the substance, all discussion regarding the pharmacology of it is purely based on its structure and subjective effect similarities to other piperidine dissociatives such as diphenidine and methoxphenidine.

In terms of its chemistry, ephenidine is a molecule of the diarylethylamine class. It contains a substituted phenethylamine skeleton with an additional phenyl ring bound to R_α. An ethyl chain is bound to the terminal amine R_N of the phenethylamine. Ephenidine is structurally analogous to diphenidine and MXP, but is not a piperidine dissociative. Ephenidine shares a diphenylethylamine skeleton with diphenidine and MXP, but lacks a piperidine substitution. 

In terms of its pharmacology, no formal studies have been carried out. However, ephenidine is thought to act as an NMDA receptor antagonist due to its structural similarities to diphenidine. NMDA receptors allow for electrical signals to pass between neurons in the brain and spinal column; for the signals to pass, the receptor must be open. NMDA receptor antagonists close the NMDA receptors by blocking them. This disconnection of neurons leads to loss of feeling, difficulty moving, and eventually the famous “hole.” This compound induces considerable stimulation at higher doses which suggests that it might function as a serotonin, dopamine and noradrenaline reuptake inhibitor in a similar manner to other dissociatives. 

The effects listed below are based upon the subjective effects index and personal experiences of PsychonautWiki contributors. The listed effects will rarely (if ever) occur all at once, but heavier dosages will increase the chances and are more likely to induce a full range of effects.

Physical effects:

The subjective physical effects of ephenidine can be broken down into several components which progressively intensify proportional to dosage. These are described below and generally include:

• Tactile disconnection
• Spontaneous tactile sensations - The ephenidine body high is a soft and pleasurable tingling sensation which is motionless and all-encompassing with no specific location.
• Stimulation - This compound provides stimulating effects at lower dosages which are less intense than that of diphenidine or methoxphenidine.
• Tactile suppression - This partially to entirely suppresses one’s sense of touch, creating feelings of numbness within the extremities. It is responsible for the anaesthetic properties of this substance.
• Motor control loss - A loss of gross and fine motor control alongside of balance and coordination is prevalent within the ephenidine experience and becomes especially strong at higher doses. This means that one should be sitting down before the onset (unless one is experienced) in case of falling over and injuring oneself.
• Decreased bodily weight - This creates the sensation that the body is floating and has become entirely weightless. This effect is strangely stimulating and encourages physical activities at low to moderate doses by making the body feel light and effortless to move.
• Physical autonomy
• Increased heart rate

Cognitive effects:

The general head space of ephenidine is often described as particularly euphoric and clear-headed in comparison to that of DXM and ketamine. The specific cognitive effects can be broken down into several separate subcomponents which are listed and described below:

• Depersonalization
• Derealization
• Consciousness disconnection
• Memory suppression (ego death)
• Thought deceleration
• Information processing suppression
• Time distortion
• Euphoria
• Introspection
• Déjà vu
• Conceptual thinking
• Compulsive redosing
• Anxiety suppression
• Disinhibition
• Amnesia
• Unity and interconnectedness

Visual effects:

Suppressions:

This substance does not enhance visual stimuli; instead, it tends to degrade and decrease visual aptitude in a variety of ways which generally include:

• Visual disconnection - This eventually results in the ephenidine equivalent of the famous “k-hole” or, more specifically, holes, spaces and voids alongside of structures. However, it is worth noting that particularly heavy dosages must be consumed to reach the deepest state of this component in comparison to other more classical dissociatives such as ketamine or methoxetamine.
• Visual acuity suppression
• Double vision - This component is prevalent at moderate to heavy dosages and makes reading impossible unless one closes an eye.
• Pattern recognition suppression - This effect generally occurs at higher dosages and makes one unable to recognize and interpret perceivable visual data.

Distortions:

Ephenidine exhibits a full array of dissociative distortions and alterations in visual perception which generally includes:

• Drifting (melting, flowing, breathing and morphing) - In comparison to other dissociatives, this effect is more prominent than ketamine, MXE, diphenidine and methoxphenidine. The visual drifting is simplistic, slow and smooth in motion, static in appearance and unrealistic/cartoon-like in style.
• Perspective distortions
• Scenery slicing

Geometry:

The visual geometry found within ephenidine can be described as very distinct and psychedelic when compared to that of ketamine, MXE, methoxphenidine and diphenidine. It is considerably less detailed than that of DXM. It does not extend beyond level 5 and can be comprehensively described through its variations as simplistic in complexity, algorithmic in style, synthetic in feel, unstructured in organization, dimly lit in lighting, multicoloured in scheme, glossy in shading, soft in edges, small in size, slow in speed, smooth in motion, equal in rounded and angular corners, immersive in depth and consistent in intensity.

Hallucinatory states:

At high doses, ephenidine can produce a full range of high level hallucinatory states in a fashion that is less consistent and reproducible than that of many other commonly used psychedelics. These effects include:

• Internal hallucinations (autonomous entities; settings, sceneries, and landscapes; alterations in perspective and scenarios and plots) -  In comparison to other dissociatives, this effect can occur at heavy dosages, but is considerably less common than the same effect found within psychedelics and deliriants. It can be comprehensively described through its variations as delirious in believability, fixed in style, equal in new experiences and memory replays in content, autonomous in controllability and solid in style.

Auditory effects:

The auditory effects are common in their occurrence and exhibit a range of effects which commonly includes:

• Enhancements
• Suppression
• Distortions
• Hallucinations

Physical health effects, potential addiction and tolerance:

The toxicity and long-term health effects of recreational ephenidine use have not been studied in any scientific context and the exact toxic dosage is unknown. This is because ephenidine is a research chemical with very little history of human usage. Anecdotal evidence from people who have tried ephenidine within the psychonaut community suggests that there are no negative health effects attributed to simply trying this drug at low to moderate doses or using it very sparingly (but nothing can be completely guaranteed).

There is a very strong tolerance build up with ephenidine which results in the need to consume increasingly large doses in order to achieve the same level of effects. This should reset to baseline after 1 - 2 weeks. There may be some addictive potential, but this is still unknown.

Legal issues:

Ephenidine is currently a legal grey area drug worldwide and is freely available through the use of online research chemical vendors. However, this does not guarantee anyone to be immune from legal prosecution should they be found in possession of this substance as the legality is likely to vary from country to country.

Conclusion:

In conclusion, ephenedine is a deeply profound dissociative which presents a full range of effects at high enough doses. Although it is not an MXE replacement, it is perhaps the best novel dissociative to recently arrive on the legal research chemical scene and is superior to that of diphenidine or MXP in my opinion.

This substance breakdown was written through collaboration between PsychonautWiki contributors Kaylee, PJosepherum, Josikins and Oscarette. The full article is available here.

Introduction:

The guide below is a recipe for extracting homemade DMT using freely accessible ingredients which can be purchased in stores or online. It is provided for informational and educational purposes only. We do not encourage you to break the law and are not responsible for your actions.

This DMT extraction was written by PsychonautWiki administrator Kaylee before being edited and proofread by Oscarette. For more information and larger pictures of this extraction process, please see our full article here. For information on DMT and its effects, please click here.

Objective:

Acacia confusa root bark contains up to 1.15% DMT/N,N-Dimethyltryptamine. ^[1] If the steps of this extraction are followed precisely on 500g of acacia root bark, you can expect a maximum theoretical yield of 5.75g of DMT. Experimental yields are highly dependent on adherence to procedure, hence this extraction can produce a range of 0g - 4.5g. It is recommended to run a test extraction on a lesser amount of acacia confusa root bark, such as 200g. With experience, as one becomes familiar with the process, the extraction will produce higher yields. 

Safety:

• Naphtha (DANGER: Extremely flammable) - Naphtha is a petroleum solvent comparable in strength to paint thinner, but more volatile. Naphtha has a low flash point, making it highly flammable; it behaves similarly to lighter fluid. Naphtha is harmful if it is inhaled, swallowed, or if the liquid is aspirated into the lungs. Naphtha is an irritant to the eyes and skin. In the event of skin contact, washing your hands with soap will prevent irritation. Excessive inhalation can cause negative health effects. Naphtha can be handled safely, but should not be inhaled or swallowed. To prevent exposure, allow the DMT to fully dry. Naphtha can be found in USA hardware or paint stores (e.g. VM&P Naphtha.) ^[2]

• Sodium Hydroxide (also known as lye or caustic soda) - Sodium hydroxide is very hazardous in case of skin or eye contact, ingestion, or respiration. Eye contact can result in corneal damage or blindsides while skin contact can produce inflammation and blistering. Exposure can result in chemical burns because sodium hydroxide solutions can readily decompose proteins and lipids in body tissues via hydrolysis. Exposure will leave the skin feeling soapy and will not immediately damage the skin. Wash hands thoroughly if this soapy sensation or irritation is felt. Wear gloves and protective eye wear when handling this substance. Repeated or prolonged exposure can be toxic. When sodium hydroxide is added to the glass jar, it must be done gradually to prevent the strong exothermic reaction that will crack the container. Sodium hydroxide is deliquescent and readily absorbs moisture from the air, so you should keep the container sealed. Sodium hydroxide can be found in USA hardware stores sold as Red Devil Lye. ^[3]

Theory:

This extraction uses a traditional acid-base methodology to extract freebase DMT. The first steps (the acid wash using vinegar and water) acidify the solution to a pH of 4. Vinegar reacts with the DMT in the plant material to create DMT acetate; this stage is often called the de-fat. Converting DMT to a salt form allows for easier absorption by the polar solution. Basifying the solution to a pH of 13 in the following steps allows it to be extracted from the polar solution (water) to the nonpolar solution (naptha). The basic solution reacts with DMT acetate through deprotonation to produce the pure basic form of DMT (the freebase). A high pH solution reduces solubility of DMT. When naphtha is added, the DMT transfers into this nonpolar solution layer. DMT is virtually insoluble in this aqueous phase, so it precipitates out of the naphtha solution via crystallization when kept at freezing temperatures.

Reagents/Materials:

• Naptha
• Lye/Sodium Hydroxide (NaOH)
• Acacia confusa root bark (shredded)
• Vinegar
• 2L glass container
• 2 large pots
• Turkey baster (glass)
• Chemical resistant gloves
• Safety goggles
• Stove

Optional:

• Funnel
• PH meter

Note: The measurements given are meant for an extraction using 500g acacia confusa root bark. They can be scaled up or down if running an extraction on a different amount of root bark. One can adjust the quantity of reagents used in this extraction proportionally with the amount of root bark used. For example, to extract 100g of root bark one would scale the proportions of the other materials accordingly to 20% of their original amounts. Do not scale down duration in steps such as cooking time in step 2; only scale the quantity of reagents. 

Preparation:

If possible, purchase shredded acacia confusa root bark. If you cannot obtain this, use a coffee grinder or blender to shred the bark into fine pieces. However, a coffee grinder will not be sufficient for large root bark pieces. Hence it is advisable to purchase the bark shredded. Using shredded bark has a large impact on extraction yields. Finely shredded or powdered bark results in higher surface area for the chemical reactions to occur.

Freeze the root bark, then let it thaw out. Repeat this three times. Freezing and thawing causes the plant cells to undergo lysis. Lysis is the destruction of the cell wall and membrane, resulting in the disintegration of the cell. Decomposing the cell membrane allows for higher absorption of DMT from the plant matter in the following steps. 

Procedure:

1. In a large pot (pot A), add 1800mL of water and 200mL of vinegar. This brings the pH of the solution to approximately 4. This will convert the DMT in the plant material in its acid salts (which are soluble in water). Most other alkaloids from the plant material are not soluble and will not be absorbed by the solution. This step helps remove impurities by separating the DMT from other plant alkaloids. These other alkaloids are not physically harmful, but lower purity results in an increased body load. Add the 500g of root bark, then boil with a lid on the pot for at least an hour and 30 minutes. This gives sufficient time to separate the DMT from the plant material.

2. Strain the solution into another large pot (pot B). Refill pot A with 1800mL of water and 200mL of vinegar, then add the root bark back into pot A. Boil for another cycle of an hour and 30 minutes. The desired end result is 1000mL (but some excess is fine).

3. Repeat step 2 one more time. Repeatedly transferring the solution to pot B and adding fresh water and vinegar to pot A encourages increased reaction of DMT with the solution. This helps prevent the reaction from reaching equilibrium with the solution, causing more DMT to react out of the plant material.

4. Now, reduce the contents of pot B (a total of somewhere around 4000mL) down to 1200mL by boiling without a lid. The liquid should now be a rusty red color while it is hot. This process will take approximately 40 minutes. Reducing the volume of liquid at this stage increases the concentration of DMT within the solution by evaporation. This serves to make the following steps easier by requiring less reagents.

5. Allow the liquid in pot B to cool, then pour it into a 2L glass container and allow it to completely cool in the refrigerator. This will take at least two hours. Allowing the liquid to cool helps prevent excess heat from the sodium hydroxide reaction from damaging the jar in the following steps.

6. For the following steps, take appropriate safety precautions and wear safety goggles and chemical resistant gloves. Sodium hydroxide can cause chemical burns and blindness. Maximum care should be taken while handling.

7. Weigh out 120 grams of sodium hydroxide. Slowly add it in small increments to the solution in the glass container. The contents of the container will heat up rapidly with the addition of the sodium hydroxide. It is important to add it slowly to prevent the glass container from exploding from the exothermic reaction. A safe guideline is adding 20g every 2 minutes and stirring in between. The contents will turn grey then black as the pH increases. This step aims to reach a pH of 13; at this pH level, the solution will be black. This step converts DMT into its freebase form, which can be extracted with naphtha.

7a. (Optional) Measure the pH of the solution with a pH meter. For ideal results, the solution should reach a pH of 13.

8. The contents of the jar should still be warm from the previous step. If not, double boil the jar to warm the solution. To do this, fill a large pot with water and bring it to a boil. Now, hold the jar suspended in the boiling water. Keep the jar suspended in the boiling water so that it doesn’t touch the bottom of the pot. A double boil allows even heat distribution by heating through contact with the water surrounding the jar instead of heating it through contact with the bottom of the pot. Then add 500mL of naphtha to the jar and seal it.

9. Shake vigorously for 3 minutes and then let the contents separate for 10 minutes. Repeat this step 4-5 times, keeping the jar contents warm. Shaking the jar encourages maximum dispersion of its contents; this allows more DMT freebase to be pulled into the naphtha layer.

10. Now allow the naphtha layer to separate fully. This may take up to several hours.

11. Using a glass turkey baster, carefully transfer the top naphtha layer into an appropriately sized crystallization jar. Avoid transferring any of the black basic layer. Error on the side of caution as it is preferable to prevent contamination by leaving the remnant of the naphtha layer. It can be difficult to prevent contamination as the naphtha layer left in the solution jar is transferred. A wide mouth jar for crystallization will be easier to scrape the DMT from.

12. Place the jar into the freezer for at least 48 hours. The DMT will gradually precipitate out of the naphtha solution. 72 hours gives ample time for it to crystallize completely. DMT is insoluble in the naphtha solution at low temperatures so it precipitates out. If the naphtha solution is left at room temperature before the DMT is separated from it, the DMT can potentially dissolve back into the naphtha.

13. To separate the DMT from the naphtha solution, pour the contents of the crystallization jar through a funnel with a pipe screen or coffee filter. Allow the extracted DMT to dry for ten hours. It will dry into powdery crystal or a waxy consistency based on the purity of the extraction. The remaining naphtha can be reused in additional pulls. Steps 7-13 can be repeated about 5 times for decent yields. A pull can result in 200mg-1g of DMT dependent on the success of the extraction. Using pH strips and accurate/sufficient equipment is beneficial to increase yields. 

Dosage (oral)
Threshold: < 20 µg
Light: 25 - 75 µg
Common: 50 – 150 µg
Strong: 150 – 400 µg
Heavy: > 400 µg

Duration (oral)
Total duration: 8 - 10 hours
Onset: 30 minutes - 2 hours
Peak: 2 - 5 hours
Come down: 2 - 4 hours
After effects: > 2 hours

1-propionyl-lysergic acid diethylamide (abbreviated as 1P-LSD or 1P-LAD) is a hallucinogenic psychedelic drug of the lysergamidefamily.

This substance has little to no history of human usage. It has not been reported upon within any formal scientific literature and is almost entirely unknown by both the academics and public. However, given its similarity to LSD and ALD-52, the structural similarity reliably suggests an extremely similar effect profile. Due to the lack of research regarding the substance, all discussion regarding the pharmacology of it is purely based on its structure and subjective effect similarities to other lysergamides.

Over the first few months of 2015, it came to be marketed as a legal alternative to LSZ, AL-LAD and LSD through online research chemical vendors which are currently selling the compound in the form of freely available blotter sheet tabs. It is usually marketed as a research chemical which means that it is sold legally but not meant for human consumption.

In terms of its chemistry, 1P-LSD is a member of the lysergamide family and is generally regarded as a psychedelic. It is analogous to LSD, featuring a propionyl group bound to the nitrogen of the indole group. It is homologous to ALD-52, which contains an acetyl group instead of the propionyl group bound to the same location.

In terms of its pharmacology, 1P-LSD acts as a 5-HT2A partial agonist. The psychedelic effects are believed to come from 1P-LSD’s efficacy at the 5-HT2A receptors. However, the role of these interactions and how they result in the psychedelic experience continues to remain elusive.

It has been theorized that 1P-LSD may be a prodrug to LSD. Despite this prevailing theory, however, it appears that this is likely untrue. It has both a shorter duration, subtly distinct subjective effects and an extremely close potency which are three things which are rarely associated with a prodrug (especially in combination).

The effects of 1P-LSD are almost identical to that of its close structural relative LSD with the differences being so minuscule they are almost negligible or primarily due to different responses within the tripper.

The effects listed below are based upon the subjective effects index and personal experiences of PsychonautWiki contributors. The listed effects will rarely if ever occur all at once but heavier dosages will increase the chances and are more likely to induce a full range of effects.

Physical effects:

The physical effects of 1P-LSD can be broken down into several components all of which progressively intensify proportional to dosage. These are described below and generally include:

• Spontaneous tactile sensations - The “body high” of 1P-LSD can be described as proportionally very intense in comparison to its accompanying visual and cognitive effects. It behaves as a euphoric, fast moving, sharp and location specific tingling sensation. For some, it is manifested spontaneously at different unpredictable points throughout the trip, but for most it maintains a steady presence that rises with the onset and hits its limit once the peak has been reached. At moderate to high doses of 1P-LSD, this sensation will usually hit its highest level and become so overwhelming that people may find themselves writhing on the floor in complete pleasure.
• Stimulation - In terms of its effects on the physical energy levels of the tripper, 1P-LSD is usually considered to be very energetic and stimulating without being forced. For example, when taken in any environment it will usually encourage physical activities such as running, walking, climbing or dancing. In comparison, other more commonly used psychedelics such as psilocin are generally sedating and relaxed.
• Nausea - Mild nausea is occasionally reported when consumed in moderate to high dosages and either passes instantly once the tripper has vomited or gradually fades by itself as the peak sets in.
• Tactile enhancement
• Bodily control enhancement
• Salivation
• Increased heart rate
• Pupil dilation

Cognitive effects:

The cognitive effects of 1P-LSD can be broken down into several components all of which progressively intensify proportional to dosage. In comparison to other psychedelics such as psilocin, LSA and ayahuasca, 1P-LSD is significantly more stimulating and fast paced in terms of the specific style of thought stream produced and contains a large number of potential effects.

The most prominent of these cognitive effects generally include:

• Current mind state enhancement
• Thought acceleration
• Novelty enhancement
• Time distortion
• Analysis enhancement
• Personal bias suppression
• Conceptual thinking
• Memory suppression (ego death)
• Thought loops
• Feelings of interdependent opposites
• Delusions
• Unity and interconnectedness
• Feelings of self-design
• Spirituality enhancement
• Delineation of thought

Visual effects:

Enhancements

1P-LSD presents a full and complete array of visual enhancements which generally includes:

• Visual acuity enhancement
• Colour enhancement
• Pattern recognition enhancement

Distortions

1P-LSD presents a full and complete array of visual distortions which generally includes:

• Drifting (melting, breathing, morphing and flowing) - In comparison to other psychedelics, this effect can be described as highly detailed and cartoon-like in its appearance. The distortions are slow and smooth in motion and fleeting in their appearance.
• Tracers
• Depth perception distortions
• Symmetrical texture repetition
• Colour shifting
• Perspective distortions

Geometry

The visual geometry that is present throughout this trip can be described as more similar in appearance to that of 2C-B or 2C-I than psilocin, LSA or DMT. It can be comprehensively described through its variations as primarily intricate in complexity, algorithmic in form, unstructured in organization, brightly lit, colourful in scheme, synthetic in feel, multicoloured in scheme, flat in shading, sharp in edges, large in size, fast in speed, smooth in motion, angular in its corners, non-immersive in depth and consistent in intensity.

In the case of higher level geometry, this substance is level 8A dominant but is also capable of inducing 8B Geometry under the right circumstances.

Hallucinatory states

1P-LSD is capable of producing a full range of low and high level hallucinatory states in a fashion that is significantly less consistent and reproducible than that of many other commonly used psychedelics. These effects include:

• Transformations
• Internal hallucinations (Autonomous entities, Settings, sceneries, and landscapes, Alterations in perspective and Scenarios and plots) - Although 1P-LSD is technically capable of producing hallucinatory states in a fashion that is on par with psilocin or DMT in its vividness and intensity, these effects are rarer and more inconsistent in comparison. While traditional psychedelics such as LSA, ayahuasca and mescaline will induce internal hallucinations near consistently at level 5 geometry and above, 1P-LSD will for most simply go straight into Level 8A visual geometry. This lack of consistently induced hallucinatory breakthroughs means that for most, 1P-LSD is not quite as deep of an experience as certain other psychedelics. On the occasion that they are induced, however, they can be comprehensively described in terms of their variations as lucid in believability, interactive in style, new experiences in content, autonomous in controllability and geometry-based in appearance.

Auditory effects:

The auditory effects of 1P-LSD are common in their occurrence and exhibit a full range of effects which commonly include:

• Enhancements
• Distortions
• Hallucinations

Physical health effects, potential addiction and tolerance:

The toxicity and long term health effects of recreational 1P-LSD use do not seem to have been studied in any scientific context and the exact toxic dosage is unknown. This is because 1P-LSD is a research chemical with very little history of human usage. Anecdotal evidence from people within the psychedelic community who have tried 1P-LSD suggests that there are no negative health effects attributed to simply trying this drug at low to moderate doses or using it very sparingly (but nothing can be completely guaranteed).

1P-LSD is non-habit forming and the desire to use it can actually decrease with use. It is most often self-regulating.

An almost immediate tolerance is built to 1P-LSD after ingestion, preventing one from experiencing its full effects more often than every 4-7 days unless they increase their dose significantly.

Legal issues:

1P-LSD is currently a grey area compound within all parts of the world. This means that it is not known to be specifically illegal within any country, but that people may still be charged for its possession under certain circumstances such as under analogue laws and with the intent to sell or consume.

Conclusion:

In conclusion, 1P-LSD is an exciting new research chemical with little to no information regarding its existence within the formal scientific literature. In terms of the effects it produces, it is identical to LSD apart from a slightly shorter duration and is therefore an excellent and worthwhile alternative which is superior to that of LSZ and AL-LAD.

This substance breakdown was written through collaboration between PsychonautWiki contributors Josikins and Oscarette. The full article (including references) is available here.

Dosage (oral)
Threshold : 5 - 10 mg
Light : 10 – 30 mg
Common : 30 – 50 mg
Strong : 50 – 70 mg
Heavy : 70+ mg

Duration (oral)
Total duration : 4 - 6 hours
Onset  : 1 - 3 hours
Peak : 2 - 3 hours
Come down : 1 - 2 hours
After effects : > 2 hours

2C-C (2,5-Dimethoxy-4-chlorophenethylamine) is a psychedelic phenethylamine of the 2C-x family.^[1] It was first synthesized by Alexander Shulgin and described in his 1991 book PiHKAL: A Chemical Love Story.

In modern times, it is used as a recreational drug and an entheogen, rarely sold on the streets and almost exclusively obtained as a grey area research chemical through the use of online vendors. Therefore, it is relatively uncommon and has only a short history of human use.

Many users report that 2C-C is gentler and more sedating than other closely related psychedelic phenethylamines.

In terms of its chemistry, 2C-C is a substituted phenethylamine with methoxy groups attached to carbons R₂ and R₅ as well as a chlorine attached to carbon R4.

In terms of its pharmacology, 2C-C’s psychedelic effects are believed to come from its efficacy at the 5-HT_2A receptor as a partial agonist. However, the role of these interactions and how they result in the psychedelic experience continues to remain elusive.

The effects listed below are based upon the subjective effects index and personal experiences of PsychonautWiki contributors. The listed effects will rarely if ever occur all at once but heavier dosages will increase the chances and are more likely to induce a full range of effects.

Physical effects:

The physical effects of 2C-C can be broken down into 9 components all of which progressively intensify proportional to dosage. These are described below and generally include:

• Spontaneous tactile sensations - The “body high” of 2C-C can be described as a pleasurable, warm, soft, all-encompassing and mild tingling sensation. This maintains a consistent presence that steadily rises with the onset and hits its limit once the peak has been reached.
• Stimulation and Sedation - In terms of its effects on the physical energy levels of the tripper, the 2C-C experience begins with mild stimulation, but usually becomes very sedating as the experience progresses and lacks the distinctive energetic feeling associated with most phenethylamines. This feeling was accurately described in Alexander Shulgin’s PiHKAL as “an intense form of relaxation.”
• Nausea - Although nausea remains present, in comparison to other phenethylamines such as 2C-E, 2C-I or 2C-B, this remains extremely mild and dissipates quickly even with higher dosages. It is perhaps one of the most physically underwhelming psychedelics out there for this reason alone.
• Bodily control enhancement and Motor control loss - Whilst at lower doses one may find that their bodily control is enhanced, at higher dosages it seems to become suppressed.
• Tactile enhancement
• Temperature regulation loss
• Salivation
• Increased heart rate

Cognitive effects:

The head space of 2C-C is described by many as one which is both insightful and relatively normal in its thought processes even at moderate to high dosages.

The total sum of these cognitive components regardless of the setting generally includes:

• Analysis enhancement
• Empathy, love and sociability enhancement - This component is consistently manifested only in the context of social settings in which one is within the company of others. These feelings of sociability, love and empathy are a little weaker and less sharp than those found on substances such as MDMA and 2C-B. They primarily manifest during the stimulating onset of the experience and dissipate once the full extent of the trip takes place.
• Thought acceleration
• Time distortion
• Novelty enhancement
• Sexual arousal
• Conceptual thinking
• Thought connectivity
• Current mind state enhancement
• Personal bias suppression
• Memory suppression
• Unity and interconnectedness

Visual effects:

Enhancements

2C-C presents a full and complete array of possible visual enhancements which generally includes:

• Acuity enhancement
• Colour enhancement
• Pattern recognition enhancement

Distortions

2C-C presents a full and complete array of possible visual distortions which generally includes:

• Drifting (melting, flowing, breathing and morphing) - In comparison to other psychedelics, this effect can be described as highly detailed, slow and smooth in motion, static in appearance and realistic in style.
• Tracers
• Colour shifting

Geometry

The visual geometry that is present throughout this trip can be described as more similar in appearance to that of 4-AcO-DMT or Ayahuasca than that of LSD, 2C-B or 2C-I. They can be comprehensively described as structured in their organization, organic in geometric style, intricate in complexity, large in size, slow and smooth in motion, colourful in scheme, glossy in colour, equally blurred and sharp in their edges and equally rounded and angular in their corners. They give off a contradictory natural and synthetic feel to them that at higher dosages. While the geometry for 2C-C has yet to be formally confirmed, it seems more likely to result in states of Level 8B visual geometry over Level 8A.

Hallucinatory states

2C-C produces a full range of high level hallucinatory states in a fashion that is more consistent and reproducible than that of many other commonly used psychedelics. This holds particularly true in comparison to other substances within the phenethylamine family. These effects include:

• Transformations
• Internal hallucinations - In comparison to other psychedelics such as LSD, 2C-C is extremely high in hallucinations embedded within visual geometry. This particular effect commonly contains hallucinations with scenarios, settings, concepts and autonomous entity contact. They are more common within dark environments and can be described as internal in their manifestation, lucid in believability, interactive in style and almost exclusively of religious, spiritual, mystical or a transcendental nature in their overall theme.
• External hallucinations - 2C-C is capable of external hallucinations embedded within visual geometry. This particular effect commonly contains hallucinations with scenarios, settings, concepts and autonomous entity contact. They can be described as external in their manifestation, lucid in believability, interactive in style and almost exclusively of religious, spiritual, mystical or a transcendental nature in their overall theme.
• Machinescapes

Auditory effects:

The auditory effects of 2C-C are common in their occurrence and exhibit a full range of effects which commonly include:

• Enhancements
• Distortions
• Hallucinations

Physical health effects, potential addiction and tolerance:

The toxicity and long term health effects of recreational 2C-C use do not seem to have been studied in any scientific context and the median lethal dose (LD50) is unknown. This is because 2C-C is a research chemical with very little history of human usage. Anecdotal evidence from those who have used 2C-C would suggest that there are little to no immediate or short-term negative health effects attributed to simply trying the drug at low to moderate doses or using it very sparingly. The validity of these statements, however, cannot be confirmed.

The addictive potential of 2C-C has not yet been formally studied, but anecdotal evidence would suggest that 2C-C is not physically addictive. Many users describe a self-regulating quality and commonly report a tolerance build up when using the drug consecutively many days in a row. A build up in tolerance may lead to weakened effects and a diminished experience, however, this effect is nearly non-existent when consumption periods are spaced 5-7 days apart.

Legal issues:

• United States: 2C-C is listed in schedule I of section 202© of the Controlled Substances Act in the United States. This was signed into law as of July 2012 under the Food and Drug Administration Safety and Innovation Act.
• United Kingdom: 2C-C is a class A controlled substance as it is covered by the phenethylamine derivatives clause of the Misuse of Drugs Act of 1971.
• Sweden: 2C-C is classified as a “health hazard” as of March 1, 2005 in their regulation SFS 2005:26, making it illegal to sell or possess.
• Japan: 2C-C is controlled by the Pharmaceutical Affairs Law in Japan, making it illegal to possess or sell.
• Australia: Australia has a blanket ban over all substituted phenethylamines including the entire 2C-X family.

Conclusion:

Despite its lack of relative potency, 2C-C definitely holds up as an excellent psychedelic and is particularly useful for those who are inexperienced. Although I have yet to determine the proper dosage for this method, I would highly recommend smoking or vaporizing this compound using a standard water bong (like one used to smoke cannabis) as it delivers an intense experience which is somewhat on par with a phenethylamine-esque DMT breakthrough experience.

This substance breakdown was written through collaboration between PsychonautWiki contributors PJosepherum, Josikins and Oscarette. The full article, including references, is available here.

Dosage (oral)
Threshold : 50 mg
Light : 70 – 85 mg
Common : 85 – 110 mg
Strong : 110 – 150 mg
Heavy : >150 mg

Duration (oral)
Total duration : 2 - 5 hours
Onset  : 15 - 30 minutes
After effects : 4 - 24 hours

Diphenidine (1-(1,2-diphenylethyl)piperidine) is a dissociative NMDA receptor antagonist and anesthetic drug that has been sold online as a research chemical.

The synthesis of diphenidine was first reported in 1924 and employed a nitrile displacement reaction analogous to the one that would later be used to discover phencyclidine in 1956. Shortly after the 2013 UK ban on arylcyclohexylamines, diphenidine and the related compound methoxphenidine became available on the grey market. Anecdotal reports describe high doses of diphenidine producing “bizarre somatosensory phenomena and transient anterograde amnesia.”

In 2014, there have been two published reports of diphenidine being sold in combination with synthetic cannabinoids in Japanese herbal incense blends. A herbal incense sold in the Shizuoka Prefecture under the name “Aladdin (sic) Spacial Edition” was found to contain diphenidine and 5-fluoro-AB-PINACA at concentrations of 289 mg/g and 55.5 mg/g, respectively. Another product called ‘‘Herbal Incense. The Super Lemon’’ containing AB-CHMINACA, 5F-AMB, and diphenidine was implicated in a fatal overdose.

Based on collaborative anecdotal evidence, diphenidine seems to have a much more rapid onset and lower half life when vaporized or smoked. When consumed this way, it is a suspected to be carcinogenic when excess heat is used. Some user reports have concluded that vaporization requires as low as 20% of what would be a common oral dose for that person.

In terms of its chemistry, diphenidine is a diarylethylamine related to methoxphenidine. It is named for its two phenyl and piperidine groups. Diphenidine’s crystalline structure is very similar to MK-801 (Dizocilpine) and shares many of the same effects. These two compounds are homeomorphs. Diphenidine is synthesized in a reaction analogous to the synthesis of PCP. The ®-enantiomer of diphenidine has a remarkable high affinity as a NMDA receptor channel blocker.

In terms of its pharmacology, diphenenidine acts as an NMDA receptor antagonist. NMDA receptors allow for electrical signals to pass between neurons in the brain and spinal column; for the signals to pass, the receptor must be open. NMDA receptor antagonists close the NMDA receptors by blocking them. This disconnection of neurons leads to loss of feeling, difficulty moving, and eventually the famous “hole”.

Although vendors of diphenidine have stated the compound acts as a dopamine-reuptake inhibitor and a serotonin reuptake inhibitor with µ-opioid affinity and typical dissociative effects, to date diphenidine has not been screened for affinity at the dopamine transporter. If this is indeed the case, however, it provides an explanation for its euphoric and often stimulating effects.

Diphenidine and related diarylethylamines have been studied in vitro as treatments for neurotoxic injury. Diphenidine may be a stronger NMDA receptor antagonist for neurogenis and neurological repair than other more common NMDA receptor antagonistic dissociatives such as ketamine, dextromethorphan, PCP analogs, Iboga and methoxetamine.

The effects listed below are based upon the subjective effects index and personal experiences of PsychonautWiki contributors. The listed effects will rarely if ever occur all at once but heavier dosages will increase the chances and are more likely to induce a full range of effects.

Physical effects:

The subjective physical effects of diphenidine can be broken down into nine components all of which progressively intensify proportional to dosage. These are described below and generally include:

• Tactile disconnection
• Spontaneous tactile sensations - The diphenidine body high is a sharp, pleasurable tingling sensation which is location specific to the hands, feet and head.
• Tactile suppression - This partially to entirely suppresses one’s own sense of touch, creating feelings of numbness within the extremities. It is responsible for the anaesthetic properties of this substance.
• Physical autonomy
• Motor control loss - A loss of gross and fine motor control alongside of balance and coordination is prevalent within diphenidine and becomes especially strong at higher dosages. This means that one should be sitting down before the onset (unless one is experienced) in case of falling over and injuring oneself.
• Euphoria - This results in feelings of physical euphoria which range between mild pleasure to powerful all-encompassing bliss.
• Decreased bodily weight - This creates the sensation that the body is floating and has become entirely weightless. This effect is strangely stimulating and encourages physical activities at low to moderate dosages by making the body feel light and effortless to move.
• Dizziness - Although uncommon, some people report dizziness under the influence of diphenidine.
• Nausea - It’s worth noting that high dose diphenidine trips can sometimes result in nausea and vomiting at the peak of the trip. For most people, this is surprisingly not as unpleasant as they would initially expect due to the accompanying detachment from the physical senses.

Cognitive effects:

The general head space of diphenidine is often described as particularly euphoric and clear headed in comparison to that of DXM and Ketamine. The specific cognitive effects can be broken down into 8 separate subcomponents which are listed and described below:

• Consciousness disconnection
• Ego suppression, loss and death
• Thought deceleration
• Information processing suppression
• Time distortion
• Euphoria
• Introspection
• Deja-Vu
• Conceptual thinking
• Compulsive redosing

Visual effects:

Suppression:

This substance does not enhance visual stimuli; instead it tends to degrade and decrease visual aptitude in a variety of ways which generally includes:

• Visual disconnection - This eventually results in the diphenidine’s equivalent of the famous “K-hole” or more specifically, holes, space and voids alongside of structures.
• Visual acuity suppression
• Double vision - This component is prevalent at moderate to heavy dosages and makes reading impossible unless one closes an eye.
• Pattern recognition suppression - This effect generally occurs at higher dosages and makes one unable to recognize and interpret perceivable visual data.

Distortions:

Diphenidine exhibits a full array of dissociative distortions and alterations in visual perception which generally includes:

• Perspective distortions
• Environmental cubism
• Environmental orbism
• Scenery slicing

Geometry:

The visual geometry found within diphenidine can be described as very dark and bland when compared to that of Ketamine or DXM and often consists of many tiny interlocking and woven lines. It does not extend beyond level 4 and can be comprehensively described through its variations as simplistic in complexity, algorithmic in style, synthetic in feel, unstructured in organization, dimly lit in lighting, multicoloured in scheme, glossy in shading, soft in edges, small in size, slow in speed, smooth in motion, equal in rounded and angular corners, immersive in depth and consistent in intensity.

Hallucinatory states:

At high dosages, diphenidine can produce a full range of high level hallucinatory states in a fashion that that is less consistent and reproducible than that of many other commonly used psychedelics. These effects include:

• Internal hallucinations (Autonomous entities, Settings, sceneries, and landscapes, Alterations in perspective and Scenarios and plots) - In comparison to other dissociatives, this effect can occur at heavy dosages but is considerably less common than the same effect found within psychedelics and deliriants. It can be comprehensively described through its variations as delirious in believability, fixed in style, equal in new experiences and memory replays in content, autonomous in controllability and solid in style.

Auditory effects:

The auditory effects are common in their occurrence and exhibit a range of effects which commonly includes:

• Enhancements
• Suppression
• Distortions
• Hallucinations

Physical health effects, potential addiction and tolerance:

The toxicity and long term health effects of recreational diphenidine use does not seem to have been studied in any scientific context and the exact toxic dosage is unknown. This is because diphenidine is a research chemical with very little history of human usage. Anecdotal evidence from people who have tried diphenidine within the psychonaut community suggests that there are no negative health effects attributed to simply trying this drug at low to moderate doses or using it very sparingly (but nothing can be completely guaranteed).

There is a very strong tolerance build up with diphenidine which results in the need to consume increasingly large doses in order to achieve the same level of effects. This should reset to baseline after 1 - 2 weeks. There may well be some addictive potential but this is still unknown.

Tolerance may be mitigated via preparing the citrate salt by mixing the chemical with citric acid or lemon juice, which will aid in absorption and increase effects.

Legal issues:

Diphenidine is currently a legal grey area drug worldwide and is freely available through the use of online research chemical vendors. However, this does not guarantee anyone to be immune from legal prosecution should they be found in possession of this substance as the legality is likely to vary from country to country.

Conclusion:

In conclusion, diphenidine feels almost identical to MXE with only subtle differences. It is a powerful hallucinogen and just as capable of inducing deep states of detachment which rival that of the other more commonly used dissociatives.

This substance breakdown was written through collaboration between PsychonautWiki contributors Kaylee, PJosepherum, Josikins and Oscarette. The full article, including references, is available here.

DISCLAIMER: I do not encourage you to break the law and this guide is purely for informational purposes only. I am not responsible for your actions.

Theory:

This extraction uses acid-base reactions to extract the DXM HBr from cough syrup to its finished product, DXM salt in lemon juice. The basic ammonia converts the DXM to freebase. The Naptha is used as a nonpolar solvent to extract the DXM from the ammonia. Then, the citric acid binds with the freebase DXM to produce DXM hyrdocitrate, a DXM salt soluble in water. This extraction has a yield around 90-95% when followed carefully.

Materials:

• DXM containing cough syrup. Cough syrup containing Dextromethorphan HBr ONLY is ideal. Syrups that also contain Guaifensin are acceptable. Do NOT use any syrups containing any other active ingredients, such as acetaminophen.
• Ammonia (clear, unscented)
• Naptha or Lighter Fluid
• Citric Acid or Lemon Juice

Equipment:

• Gallon sized unpleated zipper-seal bags
• Pan and stove
• A large glass bottle or container

Safety:

• Use the correct cough syrup as using a cough syrup with active ingredients other than Dextromethorphan HBr is very dangerous.
• Ammonia is flammable and irritating/caustic to the skin. It is caustic to the respiratory system if vapors are inhaled.
• Naptha is a hazardous chemical with multiple safety concerns. It is extremely flammable. It is irritating to the eyes and skin. Vapors can be irritating to the respiratory system and in high concentrations can depress the CNS.
• Do not use plastic containers with naptha (or, ideally, at all). Naptha can chemically attack and degrade many plastics.

Preparation:

If using citric acid, prepare a solution in water ahead of time. For each bottle of tussin, use 1.5 tablespoons of citric acid in 4 ounces of water. If using lighter fluid, be sure it evaporates without residue.

Procedure:

1. Add the cough syrup to the large glass bottle or sealable container.
2. Add in the same volume of ammonia (some excess is not a problem) and stir.
3. Tightly seal the container and shake vigorously for about 3 minutes.
4. Add in Naptha or lighter fluid, approximately 10% of the mixture in volume (a ½-1 inch layer) and shake for another 5 minutes.
5. Pour the liquid into a large gallon-sized zip lock container.
6. Let the mixture separate for 5-15 minutes. The resulting product should have two layers: the naptha layer (which the DXM is now bound to) on top and the ammonia-syrup layer on the bottom.
7. Cut a small hole in one corner of the zip-lock bag to drain. Dispose of the ammonia-syrup layer and collect the naptha/organic layer in a glass jar.
8. Optional: Adding 8 ounces of water to the organic naptha solution and allowing it to separate will minimize the amount of inactive ingredients (ammonia, cough syrup). Repeat this stage 1-3 times.
9. Now mix the solution with the pre-prepared citric acid solution or lemon juice. If using lemon juice, add an equal amount into the solvent layer.
10. Shake vigorously for at least 5 minutes.
11. Pour the solution into the gallon sized bag and allow it to separate, wait at least 45 minutes for the separation.
12. Now the citric acid/lemon juice layer is on the bottom with the naptha layer on top. Drain and collect the bottom layer and safely dispose of the top layer.
13. Boil the lemon juice for a few (5-10 minutes) to eliminate any left over organic solvents.
14. Allow to cool, this is your finished product.
15. Drink and enjoy!

Conclusion:

In conclusion, this simple article allows the masses to easily extract a powerful hallucinogenic substance from perfectly legal over the counter products containing DXM. This reduces the nausea associated with the compound and drastically increases potency.

The article was written and submitted by the PsychonautWiki contributor known as Kaylee. For more information and pictures of this extraction process, please see our full article here. For information on DXM and its effects, please click here.